Case ReportFever-induced ST-segment elevation in a syncopal patient with Brugada syndrome☆
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Cited by (4)
Anterior ST-Elevation in a Patient With Chest Pain and Fever
2019, Annals of Emergency MedicineCitation Excerpt :The patient in this case displayed a Brugada’s type I pattern during his acute febrile illness (Figures 1 and 3A) and reverted to a type III pattern on defervescence (Figure 2). Previous literature has demonstrated that the loss of function of a sodium-channel current, as observed in Brugada’s syndrome, is accentuated at higher temperatures and thus is more likely to trigger ventricular arrhythmias.1,2,5 Additionally, it has been shown that the prevalence of type I Brugada’s pattern is 20 times higher among patients presenting with fever than in afebrile patients.2
Induced Brugada syndrome: Possible sources of arrhythmogenesis
2017, Revista Portuguesa de CardiologiaCitation Excerpt :Associated malignant VAs can develop under most of these circumstances in BrS patients, because these individuals already have impaired cardiac ion channel function3,5,17. This association with VA and SCD is more evident with fever15,32–36, with certain drugs that are indicated to be avoided by BrS patients (Table 4)16 and energy drinks56, under vagotonic conditions9,12,13 such as a large meal21,58,59 or the recovery phase of exercise14,60,61, with hypertestosteronemia62,63 or hyperthyroidism64, and possibly with potassium imbalance65–68. Fever is recognized as a major risk factor in BrS and can even outperform drug challenge in unmasking a type 1 ECG5,28,32.
Fever-induced Brugada pattern: How common is it and what does it mean?
2013, Heart RhythmBrugada syndrome and voltage-gated calcium channels
2014, Pathologies of Calcium Channels
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Conflict of interest: no relation.